论文部分内容阅读
采用放免方法测定60例肝硬化患者,30例健康人的尿缓激肽排泄量、血浆肾素活性、血浆醛固酮水平。结果:肝硬化有腹水患者尿缓激肽排泄量显著降低;血浆肾素活性、血浆醛固酮水平显著升高。提示肾激肽释放酶一激肽系统和肾素一血管紧张素一醛固酮系统共同参与肝硬化水钠潴留及腹水的形成。
The urinary bradykinin excretion, plasma renin activity and plasma aldosterone level in 60 patients with cirrhosis and 30 healthy people were measured by radioimmunoassay. Results: Urinary bradykinin excretion was significantly decreased in cirrhotic patients with ascites; plasma renin activity and plasma aldosterone levels were significantly increased. It is suggested that the renal kallikrein-kinin system and the renin-angiotensin-aldosterone system are involved in the formation of water-sodium retention and ascites in cirrhosis.