患者男,49岁,因头晕两月入院。查体:BP 135/82mmHg(18/11kPa)。心、肺听诊未见异常,肝、脾助下均未触及,腹水征阴性,双下肢无水肿。颅脑CT检查未见异常,MRI结果为左基底节区脑梗塞。血、尿、便常规、血糖及肝、肾功能均正常,血脂 CH 94mg/dl、TG 325mg/dl,腹部 B超示轻度脂肪肝。入院后给予吡拉西坦(脑复康)6.0g/日等药物静脉输液治疗,一周后查肝功 ALT 52U/L,当时未做保肝治疗,两周后复查肝功 ALT 84U/L,分析 ALT升高可能由“脑复康”所致,随立即停用脑复康,并给予“肝泰乐、肌苷”等药物治疗。经过上述保肝治疗一周后查肝功,ALT 63U/L,两周后再次复查,ALT完全恢复正常,后又用脑复康则再次发生ALT升高。 本例患者所致ALT升高,我们分析可能为以下原因:①患者长期高脂血症,并伴有脂肪肝.由于肝细 Male patient, 49 years old, hospitalized for two months due to dizziness. Physical examination: BP 135 / 82mmHg (18 / 11kPa). Heart, lung auscultation no abnormalities, liver and spleen to help have not touched, ascites sign negative, no lower extremity edema. Brain CT examination showed no abnormalities, MRI results for the left basal ganglia infarction. Blood, urine, they routine, blood glucose and liver and kidney function are normal, blood lipids CH 94mg / dl, TG 325mg / dl, abdominal B-ultrasound showed mild fatty liver. After admission to give piracetam (Naofukang) 6.0g / day and other drugs intravenous infusion therapy, a week after the check liver function ALT 52U / L, did not do hepatoprotective treatment, two weeks after the review of liver function ALT 84U / L, Analysis of elevated ALT may be caused by “brain rehabilitation”, with the immediate withdrawal of brain rehabilitation, and given “liver Tailor, inosine” and other drug treatment. After a week after the treatment of liver protection check liver function, ALT 63U / L, two weeks after the re-examination, ALT completely returned to normal, then again with cerebral rehabilitation Kang ALT increased again. In this case, patients with elevated ALT, our analysis may be the following reasons: ① patients with long-term hyperlipidemia, accompanied by fatty liver. As the liver fine